An 60-something man complained of sudden weakness. There was no chest pain or SOB. He had normal blood pressure and perfusion and was asymptomatic at rest. He was well appearing.
An ECG was recorded:
You'll note there are P-waves.
Look at lead II across the bottom:
---There is a P-wave immediately after each T-wave (these do NOT conduct).
---There is a P-wave immediately before each QRS. Even though it appears as if that P-wave does not have enough time to conduct, the PR interval is exactly the same for every one of these, so it is very unlikely to be isorhythmic dissociation. Therefore, every other P-wave is conducting and it is thus 2nd degree AV block, Mobitz II.
Furthermore, there is a large R-wave in V1, with large S-wave in V5-V6 (RBBB) and also an axis toward aVR, implying a fascicular block as well.
We all know that there is a high incidence of progression from Mobitz II to third degree (complete) AV block, but we don't always get to see it.
Such progression to complete heart block is especially likely when there is high degree block due to disease in the conducting fibers (in contrast to the AV node alone). And the fact that there is RBBB + fascicular block shows that there is disease in these conducting fibers also.
Here only one fascicle is working, and then it is only working on every other beat!!
Management: Since these patients are at high risk of progressing to complete heart block, especially in the context of acute MI (not applicable here), it is wise to apply the external pacing pads. Get ready for emergent transvenous pacing and get the patient to an electrophysiologist who can place a permanent pacemaker.
The electrophysiologist was called. The patient remained stable.
Electrolytes, especially K, were normal. Troponin was negative.
80 minutes later, we have this ECG:
Here the ECG is annotated:
P-waves are occurring at a rate of 96 and no P-wave is conducting. The ventricular escape is 50, which is slightly faster than half of the sinus rate (96 divided by 2 = 48). So every second P-wave occurs at about the same time as the ventricular escape (you have to ignore the PVC). But since the ventricular rate is slightly FASTER than half the sinus rate, it comes a bit earlier on each beat and therefore the PR interval appears to shorten. Really they are just coincidentally coming at almost the same time.
Most isorhythmic dissociation does not also have AV block. In this case, there is isorhythmic dissociation with complete AV block.
See this post on isorhythmic dissociation without block: What is this rhythm?
An ECG was recorded:
There is a slow, wide rhythm with bizarre T-waves.What is it? What do you want to do? |
You'll note there are P-waves.
Look at lead II across the bottom:
---There is a P-wave immediately after each T-wave (these do NOT conduct).
---There is a P-wave immediately before each QRS. Even though it appears as if that P-wave does not have enough time to conduct, the PR interval is exactly the same for every one of these, so it is very unlikely to be isorhythmic dissociation. Therefore, every other P-wave is conducting and it is thus 2nd degree AV block, Mobitz II.
Furthermore, there is a large R-wave in V1, with large S-wave in V5-V6 (RBBB) and also an axis toward aVR, implying a fascicular block as well.
We all know that there is a high incidence of progression from Mobitz II to third degree (complete) AV block, but we don't always get to see it.
Such progression to complete heart block is especially likely when there is high degree block due to disease in the conducting fibers (in contrast to the AV node alone). And the fact that there is RBBB + fascicular block shows that there is disease in these conducting fibers also.
Here only one fascicle is working, and then it is only working on every other beat!!
Management: Since these patients are at high risk of progressing to complete heart block, especially in the context of acute MI (not applicable here), it is wise to apply the external pacing pads. Get ready for emergent transvenous pacing and get the patient to an electrophysiologist who can place a permanent pacemaker.
The electrophysiologist was called. The patient remained stable.
Electrolytes, especially K, were normal. Troponin was negative.
80 minutes later, we have this ECG:
Now what is going on? |
Here the ECG is annotated:
P-waves are occurring at a rate of 96 and no P-wave is conducting. The ventricular escape is 50, which is slightly faster than half of the sinus rate (96 divided by 2 = 48). So every second P-wave occurs at about the same time as the ventricular escape (you have to ignore the PVC). But since the ventricular rate is slightly FASTER than half the sinus rate, it comes a bit earlier on each beat and therefore the PR interval appears to shorten. Really they are just coincidentally coming at almost the same time.
Most isorhythmic dissociation does not also have AV block. In this case, there is isorhythmic dissociation with complete AV block.
See this post on isorhythmic dissociation without block: What is this rhythm?
Clinical Course
Regardless of whether there was progression to complete heart block or not, the patient would need a pacemaker.
A pacemaker was implanted. There was no myocardial infarction. The etiology was not yet found.
How about those T-waves??
These are common in high grade AV block. You can read more about them here:
can we get such short PR in case of conducted beat of 2nd degree AV block??? is there underlying pre-excitation or a long rhythm strip would confirm AV dissociation???
ReplyDeleteIt appears that we can!
DeleteBut any reason for such a short PR in AVB in otherwise normal PR interval ECGs?
Deletenot that I know of
DeleteLooking at the first EKG and the short PR, shouldnot we be thinking of accessort pathway?
ReplyDeleteno. there is no pre-excitation and if there were an accessory pathway, then every beat would be conducted.
DeleteThanks
DeleteThat's a great answer... 🙂
ReplyDeletewould you suspect RVH and RAE in the patient (ECG 1)... Tall R wave in V1 plus P wave morphology in V1 and lead 2 to some extend ????
ReplyDeleteIt is certainly possible, but can't be easily established in the presence of the bundle branch block
Deletehello... In ECG 2 your interpretation is isorhythmic AV dissociation..the atrial rate is 96 and ventricular rate is 50.. My question is, to call it isorhythmic AV dissociation,don't the ventricular rate and atrial has to be almost same??
ReplyDeleteIt would be a combination of isorhythmic dissociation AND second degree AV block Mobitz II. so, yes, in the presence of mobitz II, you can have isorhythmic dissociation with an atrial rate of 96 and ventricular rate of 50 because the EFFECTIVE atrial rate that you are assessing is only 48.
DeleteI'm a bit late in asking this question; however, I keep looking at the first ECG and question if there is any chance this could be an accessory pathway and U waves? You indicated the electrolytes were normal, but is there any chance that there are U waves following the T wave and not P waves? Excellent case that gets the brain going.
ReplyDeletethat might be possible until you see what happens moments later. good observation!
DeleteWhy not it u waves rather than p?
DeleteHi Dr. Smith,
ReplyDeleteI was just reviewing this for a presentation I am giving on AV blocks and I have a different take on ECG # 1 from this case. I find it hard to believe that the P waves that are right next to the QRS are conducting to the QRS directly next to it. To me this would then suggest some sort of pre-excitation. Rather, wouldn't it make more sense if that p wave is actually dropped and the p waves a bit less that mid way between the QRS is conducted (very significant AV nodal block). Marriott talks about this a lot in his books and notes PR interval can reach up to 1 second or appear to jump over a QRS ("Over the Top" in his Pearls & Pitfalls in Electrocardiography, not seen in above ECG).
Thanks!
Mike
Michael,
DeleteI think you are right about that! Good thinking! Thanks!
Steve Smith
He presets with s1q3t3, and with the T waves, and he R wave in v1, seems out of the gate I would say PE?.
ReplyDeleteNo. It's AV block.
Delete