Wednesday, June 26, 2013

K. Wang Video: ST Elevation in Conditions other than Acute STEMI

Watch this great presentation.  (For those of you who know my posts and research, you will find some slight differences in Dr. Wang's approach.)


ST Elevation in Conditions other than Acute MI from HQMedEd on Vimeo.
K. Wang, MD
Clinical Professor of Medicine
Cardiology Division
University of Minnesota

med.umn.edu/cardiology/faculty/wang/home.html

4 comments:

  1. Nice presentation !!! Though im a bit disappointed that Dr K Wang didn't show head to head Benign Repol vs Pericarditis..As i have read, PR depression is not sensitive to pericarditis..The fishhook sign is also not that sensitive for early repol...Moreover, the fishhook is actually the J point which may also present in pericarditis

    Any pearls on how to differentiate these two?

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    1. Ryan,

      here is K.'s answer: In my experience the “fishhook” is essential to make the Dx of early repolarization. I have seen very minute (a baby) “fishhook” in pericarditis but very rarely. Near the end of my session, one frame showed early repol and the next frame which had composite of 7 cases of ST elevation had pericarditis. The PR segment depression in pericarditis reflects atrial counterpart of ST elevation, i.e. subepicardial injury pattern of the atrial wall and is almost always there.
      Thanks for the comments.
      K. Wang.


      My answer would also include that pericarditis is very unusual. It also is not important to diagnosis it. If you suspect it, do a bedside ultrasound to rule out effusion and look for good pump function, and maybe checkk troponin to look for myocarditis. The main problem with pericarditis is that it can mimic STEMI, but even this is not a big problem because it is so rare. I always say: "you diagnose pericarditis at your peril." because it is more likely STEMI than pericarditis. If not STEMI, the who cares if it is pericarditis or early repol - both are benign.

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  2. Wonderfull presentation!
    Is there a way to distinguish right ventricle overload/dilation/ischaemia from acute anteroseptal STEMI based on morphology of V1-V3?

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    Replies
    1. Difficult to answer: the morphology is very recognizable subjectively, but hard to give an exact description: anteroseptal MI has slight ST elevation and upward sloping, then sharp downturn of the T-wave, whereas RV overload has a more gradual curve to the T-wave inversion. Also, the best objective way to distinguish them is the presence or absence of T-wave inversion in lead III (presence associated with RV strain).

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