tag:blogger.com,1999:blog-549949223388475481.post687062389333443752..comments2024-03-28T14:02:08.119-05:00Comments on Dr. Smith's ECG Blog: Thrombus propagation on 10 serial Prehospital ECGs: Can you explain the progression?Unknownnoreply@blogger.comBlogger7125tag:blogger.com,1999:blog-549949223388475481.post-21252501610833211452019-06-13T15:11:32.402-05:002019-06-13T15:11:32.402-05:00As per Dr. Smith — there is LAHB. Please check out...As per Dr. Smith — there is LAHB. Please check out the Answer to Question B in My Comment above — :)ECG Interpretationhttps://www.blogger.com/profile/02309020028961384995noreply@blogger.comtag:blogger.com,1999:blog-549949223388475481.post-27667191530015559502019-06-13T07:22:38.682-05:002019-06-13T07:22:38.682-05:00It is due to the new Left anterior fascicular bloc...It is due to the new Left anterior fascicular block, which creates a high voltage positive aVL.Steve Smithhttps://www.blogger.com/profile/08027289511840815536noreply@blogger.comtag:blogger.com,1999:blog-549949223388475481.post-84559378314177219352019-06-12T19:46:22.599-05:002019-06-12T19:46:22.599-05:00Great series of ECGs and discussion. Hey can I ask...Great series of ECGs and discussion. Hey can I ask question regarding the QRS voltage in aVL and inferior leads.<br />In the ECGs up to 14 minutes the voltage is tiny and T waves relatively huge in aVL. I normally look for voltage loss as sign of acute ischaemia. But after 14 mins the voltage in aVL and inferior leads gets bigger and bigger despite the obvious evolving anterior STEMI. Is this partial reperfusion of the high lateral wall while the antior wall is starting to infarct? Cheers RobAnonymoushttps://www.blogger.com/profile/13454649179425002713noreply@blogger.comtag:blogger.com,1999:blog-549949223388475481.post-56371553561426284202018-12-05T00:15:11.407-06:002018-12-05T00:15:11.407-06:00To the discussion - to me, the most alarming findi...To the discussion - to me, the most alarming finding on the first ECG is still the depression of ST segment on the inferior wall...of course the change in aVL is notable but the depression is the thing that hits the eye. Great case, good job!Candlemakerhttps://www.blogger.com/profile/18089542475369172117noreply@blogger.comtag:blogger.com,1999:blog-549949223388475481.post-29743438742417743472018-11-17T09:40:50.497-06:002018-11-17T09:40:50.497-06:00BIG THANKS professors Smith and GRAUER...BIG THANKS professors Smith and GRAUER...Hector Munozhttps://www.blogger.com/profile/10157498764409620840noreply@blogger.comtag:blogger.com,1999:blog-549949223388475481.post-75255310348606479282018-11-16T18:05:00.221-06:002018-11-16T18:05:00.221-06:00Thanks Tom! — :) KenThanks Tom! — :) KenECG Interpretationhttps://www.blogger.com/profile/02309020028961384995noreply@blogger.comtag:blogger.com,1999:blog-549949223388475481.post-41104845968240297712018-11-16T04:55:05.144-06:002018-11-16T04:55:05.144-06:00extraordinary.
the techs in my shop consider me a ...extraordinary.<br />the techs in my shop consider me a pain in the tuchus because i often order repeat ECG's. we see 72000 patients a year, and there are frequent ECG's at triage, and then i contribute to their burden in the main ER. i attempt to use discretion, but as this post illustrates, serial ECG's are an invaluable tool when used appropriately.<br /><br />excellent teaching case. once again, thank you Steve and Ken.tfierohttps://www.blogger.com/profile/15955268501222734373noreply@blogger.com