Monday, March 15, 2021

80-something year old with acute chest pain. 3 visits. Fascinating Ultrasound progression

An 80-something year old man with history of metastatic cancer had acute onset of chest pain and called 911.

Here is his prehospital ECG:

What do you think?










The computer read ***Anterior STEMI*** along with RBBB.

Smith interpretation: There is Right Bundle Branch Block (RBBB).  There is 1 mm of STE in inferior leads and also in lateral precordial leads.  As a general rule, RBBB should not have ST Elevation (there are some infrequent patients with RBBB who have non-ischemic STE, usually discordant to the negative S-wave, as in this case).  Moreover, the T-waves appear hyperacute.

I would activate the cath lab based on this and the clinical presentation, but I do NOT see any evidence of anterior ***STEMI***


The patient arrived in the ED and had this ECG: 

The T-waves are now less hyperacute, but ST Elevation remains.

A bedside echo was done:


This shows a small effusion.


The patient went to the cath lab and had no acute lesion.

He ruled out for acute MI by negative serial troponins.


Here is his ECG the next AM:

Some ST Elevation remains.  There is no typical evolution of MI (so BOTH EKG evolution, and troponin, proves there was no acute MI)



2 weeks later, the patient present with acute chest pain again.  Here is his ECG:

No STE at all, and no hyperacute T-waves.
He ruled out for MI by troponins again.


Here was his echo that evening:




Continued

9 hours later: the patient presented with fever and hypotension.

Here was his cardiac echo (remember, done 9 hours after the one above):

Pericardial Effusion with Tamponade physiology, developed within 9 hours.

Here is the ECG:

No ST Elevation


Final Diagnosis: Pericarditis

The fluid was nondiagnostic as to etiology of pericarditis.  






7 comments:

  1. Dear Sir, when he developed chest pain and there were ST depressions in V1-V3, with negative troponins before its progression to pericarditis, could it have been considered an unstable angina?. what is your take Sir?

    ReplyDelete
    Replies
    1. Well that is possible, but far more likely is pericarditis, which does explain all the findings

      Delete
  2. Is there any explanation for the voltage of inferior lead quickly diminishing after the prehospital ECG?

    ReplyDelete
    Replies
    1. Good question! But I'm not sure and it could be lead placement

      Delete
  3. Great case. I would had activated the Lab based on first ECG too. As I mentioned answering the tweet, HR seemed a little bit fast for Inferior OMI but I cannot put too much weight on it when seeing a pt with these presenting symptoms and ECG. I learned. Lot. Thank you.

    ReplyDelete
  4. Great case. I would had activated the Lab based on first ECG too. As I mentioned answering the tweet, HR seemed a little bit fast for Inferior OMI but I cannot put too much weight on it when seeing a pt with these presenting symptoms and ECG. I learned. Lot. Thank you.

    http://my-estub.bid/

    ReplyDelete
  5. Great case. I would had activated the Lab based on first ECG too. As I mentioned answering the tweet, HR seemed a little bit fast for Inferior OMI but I cannot put too much weight on it when seeing a pt with these presenting symptoms and ECG. I learned. Lot. Thank you.

    http://my-estub.bid/

    ReplyDelete

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