I was in a meeting and received a text message: "can you look at an ECG?"
I texted back to take a photo and send it.
The patient is a male over 40 years of age with central chest discomfort for several hours. Here is the ECG, as texted:
Answer
I called her back and this is what I said:
I texted back to take a photo and send it.
The patient is a male over 40 years of age with central chest discomfort for several hours. Here is the ECG, as texted:
What is your diagnosis? |
Answer
I called her back and this is what I said:
“This
is a definite posterolateral MI, with an occluded artery, probably an OM, but
it is not technically a STEMI because there is not 1 mm in 2 consecutive
leads. He needs to go to the cath lab ASAP.”
How did I know this?
Because there is BOTH: minimal STE in I and aVL with
reciprocal ST depression in III, AND ST depression in V2 and V3.
Combine this with sudden onset of substernal chest pain and
you have a certain posterolateral MI.
She called the cardiologist on call and emphasized the need to go ASAP to the cath lab. The patient's pain resolved completely with intensive medical therapy, and so the urgency was less. Within a very short time, the patient underwent an angiogram that showed a 99% OM-1 occlusion and and 85% RCA. Both appeared "hazy"; both appeared to be culprits and both underwent stenting.
A subsequent echo was normal, without wall motion abnormality. Remember this!
The troponin I peaked at 58 ng/ml, so this was not a small MI.
Learning points:
- 25-30% of "NonSTEMIs" have an occluded infarct-related artery at 24 hours after presentation. (1, 2, 3)
- Many more are occluded at the time of presentation.
- This ECG would be read as "non-diagnostic" by the vast majority of interpreters. There are many "nondiagnostic" ECGs that really are diagnostic if scrutinized closely.
- Cardiac ultrasound done after resolution of ischemia is only moderately sensitive for MI.
References
1. Wang T, Zhang M, Fu
Y, et al. Incidence, distribution, and prognostic impact of occluded culprit
arteries among patients with non–ST-elevation acute coronary syndromes undergoing
diagnostic angiography Am Heart J 2009;157(4):716-23.
2. From AM, Best PJM, Lennon RJ, Rihal CS,
Prasad A. Acute Myocardial Infarction Due to Left Circumflex Artery Occlusion
and Significance of ST-Segment Elevation. Amercan Journal of Cardiology 2010;106(8):1081-5.
3. Pride YB, et al. Angiographic and clinical outcomes among patients with ACS presenting with isolated ST-Segmment Depression: A TRITON-TIMI 38 Substudy. Journal of the American College of Cardiology: Cardiovascular Interventions 2010;3(8):806-11.
I'm curious if the lack of significant R wave progression until the low lateral leads is also indicative of posterior involvement?
ReplyDeletegood question, maybe, not sure. Hmmm.
DeleteWhy isn't this is an anterior MI based on the st segment elevation?
ReplyDeleteThere is no anterior ST segment elevation(???)
ReplyDeleteSteve -
ReplyDeleteInteresting coincidence - the AHRQ Web M&M just published and ED case regarding a missed MI. Their "ECG #1" was essentially that pictured here, with "minimal STE in I and aVL with reciprocal ST depression in III, AND ST depression in V2 and V3." Cath on visit # showed a "branch occlusion" of the LCx.
Since it's an M&M, things didn't go as well as with your patient. Check out the case at http://www.webmm.ahrq.gov/case.aspx?caseID=288, if so inclined.
Thanks for the cases - I try to review one before every shift!
Brooks Walsh
MillHillAveCommand.blogspot.com
I'm a family doc doing ER staffing. I want to be better at ekg interpretation. would you or any other cardiologist or resident you know be willing to talk through with me ekg's I get in the ER ?
ReplyDeleteYou can send me ones you're wondering about at:
Deletedr.smiths.ecg.blog@gmail.com