A woman in her 60's complained of chest pain. 911 was called. She had this prehospital ECG:
The cath lab was activated.
While waiting for the cath team to be ready, I recorded this bedside echo:
This shows excellent wall motion everywhere. I was amazed and realized that she must have had spontaneous reperfusion. (I cannot say for certain that a high quality echo with contrast would have been normal)
So I recorded an ED ECG:
Here is an enlargement of V1-V3:
So this patient had spontaneous reperfusion. She went to the cath lab and by the time she arrived, the RCA was again 100% occluded. There was ruptured plaque and thrombus. It was opened rapidly.
Peak troponin I was 0.60 ng/ml. Formal echo was normal except for a probable anterior wall motion abnormality, only seen on one view, and possibly pre-existing. EF 66%.
Here was the post cath ECG:
Lessons:
1. Spontaneous reperfusion normalizes BOTH the ECG and the echocardiogram. Only if there is persistent myocardial stunning from ischemia (which usually is present with prolonged severe ischemia) is wall motion persistently affected. If we had done an echo during the ST elevation, there would have been a wall motion abnormality, but it disappeared with reperfusion.
2. Learn this reperfusion morphology in lead V2 from reperfused posterior STEMI. It is important in recognizing ACS if you do not have a recording during pain.
3. Down-up T-waves (e.g., aVL here) is almost always a reperfusion morphology (alternatively, it can be a U-wave masquerading as a T-wave)
Obvious Infero-posterior and lateral STEMI |
The cath lab was activated.
While waiting for the cath team to be ready, I recorded this bedside echo:
This shows excellent wall motion everywhere. I was amazed and realized that she must have had spontaneous reperfusion. (I cannot say for certain that a high quality echo with contrast would have been normal)
So I recorded an ED ECG:
This is near normal, except for the abnormal T-waves (down up in aVL, and note the abnormal T-wave in V2). |
Here is an enlargement of V1-V3:
So this patient had spontaneous reperfusion. She went to the cath lab and by the time she arrived, the RCA was again 100% occluded. There was ruptured plaque and thrombus. It was opened rapidly.
Peak troponin I was 0.60 ng/ml. Formal echo was normal except for a probable anterior wall motion abnormality, only seen on one view, and possibly pre-existing. EF 66%.
Here was the post cath ECG:
Lessons:
1. Spontaneous reperfusion normalizes BOTH the ECG and the echocardiogram. Only if there is persistent myocardial stunning from ischemia (which usually is present with prolonged severe ischemia) is wall motion persistently affected. If we had done an echo during the ST elevation, there would have been a wall motion abnormality, but it disappeared with reperfusion.
2. Learn this reperfusion morphology in lead V2 from reperfused posterior STEMI. It is important in recognizing ACS if you do not have a recording during pain.
3. Down-up T-waves (e.g., aVL here) is almost always a reperfusion morphology (alternatively, it can be a U-wave masquerading as a T-wave)
I have seen common practice to discharge patients with chest pain and concerning ECGs in the setting of normal ECHO ! Is echo very sensitive in the settings of unstable angina or NSTEMI?
ReplyDeleteIn my experience, The cardiac ultrasound is only sensitive during active ischemia. That is to say, that it is sensitive when there are active ST segment and T-wave abnormalities. Once these have resolved, the echo cardiogram is no longer sensitive.
DeleteNice case
ReplyDeletein my opinion, on echo inferoseptum is slightly hypokinetic
ReplyDeleteTazky,
DeleteYou may be right. But it is not obvious.
Thanks, Steve Smith
Dr. Smith,
ReplyDeleteIf you were working in a area where a cardiac cath lab wasn't available and you had normalization of a STEMI ECG as in the case above, would there still be a place for thrombolytics?
Please keep in mind getting access to a cath lab would take more than 24 hrs.
thanks
Good question. I don't think there is a solid answer to it. However, if I were there, I think I would treat with aspirin, Plavix, heparin, and eptifibatide. I would only give thrombolytics if the ST segments rose again.
Delete